Acetylcholinesterase and inhibitors: effects upon normal and regenerating nerves of the rat
Author:
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1460-9568.1999.00510.x/fullpdf
Reference42 articles.
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3. Axonal sprouting induced in the sciatic nerve by the amyloid precursor protein (APP) and other antiproteases;Alvarez;Neurosci. Lett.,1992
4. Acetylcholinesterase peripheral anionic site degeneracy conferred by amino acid arrays sharing a common core;Barak;J. Biol. Chem.,1994
5. Peripheral axons of WldS mice, which do not regenerate, do so when transcription is inhibited in the distal stump;Benavides;Neurosci. Lett.,1998
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1. Orally administered boldine reduces muscle atrophy and promotes neuromuscular recovery in a rodent model of delayed nerve repair;Frontiers in Cellular Neuroscience;2023-09-27
2. Habituation, discrimination and anxiety in transgenic mice overexpressing acetylcholinesterase splice variants;Brain Research;2007-12
3. Astroglia up-regulate transcription and secretion of ‘readthrough’ acetylcholinesterase following oxidative stress;European Journal of Neuroscience;2006-07
4. Muscular dystrophy with laminin deficiency decreases the content of butyrylcholinesterase tetramers in sciatic nerves of Lama2dy mice;Neuroscience Letters;2002-10
5. Protein synthesis in axons and terminals: significance for maintenance, plasticity and regulation of phenotype;Progress in Neurobiology;2000-09
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