Mechanisms of decreased bradykinin- induced vasodilation in experimental hyperlipemia-hyperglycemia: contribution of nitric oxide and Ca2+-activated K+ channels-Reference-Cited by-同舟云学术

Mechanisms of decreased bradykinin- induced vasodilation in experimental hyperlipemia-hyperglycemia: contribution of nitric oxide and Ca2+-activated K+ channels

Published:2001-10-24 Issue:5 Volume:15 Page:335-342
ISSN:0767-3981
Container-title:Fundamental and Clinical Pharmacology
language:en
Short-container-title:Fundam Clin Pharmacol

Author:

Georgescu Adriana,Popov Doina,Simionescu Maya

Publisher

Wiley

Subject

Pharmacology (medical),Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1472-8206.2001.00047.x/fullpdf

Reference48 articles.

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2. Endothelium-dependent relaxation and noradrenaline sensitivity in mesenteric resistance arteries of streptozotocin-induced diabetic rats

3. Selective Impairment of Acetylcholine-Mediated Endothelium-Dependent Relaxation in Isolated Resistance Arteries of the Streptozotocin-Induced Diabetic Rat

4. Impaired endothelium-dependent vasodilation in patients with insulin-dependent diabetes mellitus.

5. Impaired Contraction and Endothelium-Dependent Relaxation in Isolated Resistance Vessels from Patients with Insulin-Dependent Diabetes Mellitus

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