Transgenic mouse model for familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation
Author:
Publisher
Wiley
Subject
Neurology (clinical),General Medicine,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1440-1789.2001.00361.x/fullpdf
Reference51 articles.
1. Superoxide dismutase-1 mutation-related neurotoxicity in familial amyotrophic lateral sclerosis
2. An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria
3. ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions
4. Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis.
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