Interferon-γ Production in Skin During Contact Hypersensitivity. No Contribution from Keratinocytes
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference5 articles.
1. Early molecular events in the induction phase of contact sensitivity;Enk;Proc Natl Acad Sci USA,1992
2. Epidermal keratinocyte production of interferon-gamma immunoreactive protein and mRNA is an early event in allergic contact dermatitis;Howie;J Invest Dermatol,1996
3. Cytotoxicity is mandatory for CD8 (+) T cell-mediated contact hypersensitivity;Kehren;J Exp Med,1999
4. Contact dermatitis I. Pathophysiology of contact sensitivity;Krasteva;Eur J Dermatol,1999
5. T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (Il) 4/Il-10-producing (Th2) negative regulatory CD4+ T cells.
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1. In vivo induction of regulatory T cells promotes allergen tolerance and suppresses allergic contact dermatitis;Journal of Controlled Release;2017-09
2. Reduced 2,4-dinitro-1-fluorobenzene-induced contact hypersensitivity response in IL-15 receptor??-deficient mice correlates with diminished CCL5/RANTES and CXCL10/IP-10 expression;European Journal of Immunology;2005-03
3. Keratinocyte Growth Inhibition by High-Dose Epidermal Growth Factor Is Mediated by Transforming Growth Factor β Autoinduction: A Negative Feedback Mechanism for Keratinocyte Growth;Journal of Investigative Dermatology;2003-06
4. Langerhans Cells Do Not Produce Interferon-γ;Journal of Investigative Dermatology;2003-05
5. Afferent and Efferent Phases of Allergic Contact Dermatitis (ACD) Can Be Induced After a Single Skin Contact with Haptens: Evidence Using a Mouse Model of Primary ACD;Journal of Investigative Dermatology;2003-04
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