Effects of [D-Ala1] Peptide T-NH2 and HIV Envelope Glycoprotein Gp120 on Cyclic AMP Dependent Protein Kinases in Normal and Psoriatic Human Fibroblasts-Reference-Cited by-同舟云学术

Effects of [D-Ala1] Peptide T-NH2 and HIV Envelope Glycoprotein Gp120 on Cyclic AMP Dependent Protein Kinases in Normal and Psoriatic Human Fibroblasts

Published:1998-04 Issue:4 Volume:110 Page:332-337
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Liapi Charis,Takahashi Noriko,Raynaud Francoise,Evain-Brion Daniele,Anderson Wayne B.

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference54 articles.

1. CC-CKR5: a RANTES, MIP-1 alpha, MIP-1 beta receptor as a fusion cofactor for macrophage-tropic HIV-1;Alkhatib;Science,1996

2. Selective modulation of protein kinase isozymes by the site selective analog 8-chloroadenosine 3′,5′-cyclomonophosphate provides a biological means for control of human colon cancer cell growth;Ally;Proc Natl Acad Sci,1988

3. Interferon in suction blister fluid from psoriatic lesions;Bjerke;Br J Dermatol,1983

4. The keratin polypeptides of psoriatic dermis;Boden;J Invest Dermatol,1978

5. Ultrastructure of the capillary loops in the dermal papillae of psoriasis;Braveman;J Invest Dermatol,1977

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Peptide T exhibits a well-defined structure in fluorinated solvent at low temperature;Journal of Peptide Science;2009-10-28

2. Chemokine receptor-5 (CCR5) is a receptor for the HIV entry inhibitor peptide T (DAPTA);Antiviral Research;2005-08

3. Regressão de psoríase em paciente HIV-positivo após terapia anti-retroviral;Anais Brasileiros de Dermatologia;2003-12

4. Update on D-Ala-Peptide T-Amide (DAPTA): A Viral Entry Inhibitor that Blocks CCR5 Chemokine Receptors;Current HIV Research;2003-01-01

5. Morphine alleviates early stages of apoptosis in cultured CEM x174 cells infected with simian immunodeficiency virus;Cell Biology International;2003