Fas-Deficient C3.MRL-Tnfrsf6lpr Mice and Fas Ligand-Deficient C3H/HeJ-Tnfsf6gld Mice Are Relatively Resistant to the Induction of Alopecia Areata by Grafting of Alopecia Areata-Affected Skin from C3H/HeJ Mice

Author:

Freyschmidt-Paul Pia,McElwee Kevin J.,Botchkarev Vladimir,Kissling Sabine,Wenzel Elke,Sundberg John P.,Happle Rudolf,Hoffmann Rolf

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,General Medicine,Biotechnology

Reference19 articles.

1. Alopecia areata: An immunologic disease?;Baadsgaard;J Invest Dermatol,1991

2. Role of cytotoxic T cells in chronic alopecia areata;Bodemer;J Invest Dermatol,2000

3. p53 Involvement in the control of murine hair follicle regression;Botchkarev;Am J Pathol,2001

4. Evidence for Fas-dependent and Fas-independent mechanisms in the pathogenesis of experimental autoimmune encephalomyelitis;Dittel;J Immunol,1999

5. Successful treatment of alopecia areata-like hair loss with the contact sensitizer squaric acid dibutylester (SADBE) in C3H/HeJ mice;Freyschmidt-Paul;J Invest Dermatol,1999

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