Overexpression of the Oncofetal Fn Variant Containing the EDA Splice-in Segment in the Dermal–Epidermal Junction of Psoriatic Uninvolved Skin-Reference-Cited by-同舟云学术

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Overexpression of the Oncofetal Fn Variant Containing the EDA Splice-in Segment in the Dermal–Epidermal Junction of Psoriatic Uninvolved Skin

Published:2000-04 Issue:4 Volume:114 Page:706-711
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Ting Kathleen M.,Rothaupt Dinah,McCormick Thomas S.,Hammerberg Craig,Chen Guofen,Gilliam Anita C.,Stevens Seth,Culp Lloyd,Cooper Kevin D.

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference40 articles.

1. Fn and alpha5 integrin regulate keratinocyte cell cycling. A mechanism for increased Fn potentiation of T cell lymphokine- driven keratinocyte hyperproliferation in psoriasis;Bata-Csorgo;J Clin Invest,1998

2. Intralesional T-lymphocyte activation as a mediator of psoriatic hyperplasia;Bata-Csorgo;J Invest Dermatol,1995

3. Kinetics and regulation of human keratinocyte stem cell growth in short-term primary ex vivo culture. Cooperative growth factors from psoriatic lesional T lymphocytes stimulate proliferation among psoriatic uninvolved, but not normal, stem keratinocytes;Bata-Csorgo;J Clin Invest,1995

4. Differential expression of adhesion molecules on infiltrating cells in inflammatory dermatoses;Boehncke;J Am Acad Dermatol,1992

5. Macrophages and fibroblasts express embryonic Fns during cutaneous wound healing;Brown;Am J Pathol,1993

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