Induction of TGF-β1 by the matricellular protein SPARC in a rat model of glomerulonephritis-Reference-Cited by-同舟云学术

Induction of TGF-β1 by the matricellular protein SPARC in a rat model of glomerulonephritis

Published:2000-01 Issue:1 Volume:57 Page:117-128
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Bassuk James A.,Pichler Raimund,Rothmier Justin D.,Pippen Jeffrey,Gordon Kathy,Meek Rick L.,Bradshaw Amy D.,Lombardi Donna,Strandjord Thomas P.,Reed May,Sage E. Helene,Couser William G.,Johnson Richard

Publisher

Elsevier BV

Subject

Nephrology

Reference56 articles.

1. Diversity of function is inherent in matricellular proteins: An appraisal of thrombospondin 1;Bornstein;J Cell Biol,1995

2. Extracellular proteins that modulate cell–matrix interactions: SPARC, tenascin, and thrombospondin;Sage;J Biol Chem,1991

3. The biology of SPARC, a protein that modulates cell–matrix interactions;Lane;Fed Am Soc Exp Biol J,1994

4. Type I collagen-deficient Mov-13 mice do not retain SPARC in the extracellular matrix: Implications for fibroblast function;Iruela-Arispe;Dev Dyn,1996

5. Thrombospondin-1 is a major activator of TGF-beta 1 in vivo;Crawford;Cell,1998

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