Differential expression of MHC class II molecules by microglia and neoplastic astroglia: relevance for the escape of astrocytoma cells from immune surveillance

Author:

Tran 1,Wolz 1,Egensperger 2,Kösel 1,Imai 3,Bise 1,Kohsaka 3,Mehraein 1,Graeber 4

Affiliation:

1. Institute of Neuropathology, Ludwig‐Maximilians‐University, Munich, Germany

2. Molecular Neuropathology Laboratory, Institute of Neuropathology, Hannover Medical School, Hannover, Germany

3. Department of Neurochemistry, National Institute of Neuroscience, Tokyo, Japan

4. Molecular Neuropathology Laboratory, Department of Neuromorphology, Max‐Planck‐Institute of Neurobiology, Martinsried, Germany

Publisher

Wiley

Subject

Physiology (medical),Clinical Neurology,Neurology,Histology,Pathology and Forensic Medicine

Reference41 articles.

1. Local invasion and intraparenchymal metastasis, of astrocytomas

2. Detection of MHC class II-antigens on macrophages and microglia, but not on astrocytes and endothelia in active multiple sclerosis lesions

3. Cellular immune response in multiple sclerosis plaques.;Boyle EA;Am J Pathol,1990

4. BurgerPC&ScheithauerBWTumours of the Central Nervous System.Atlas of Tumour Pathology (Third Series Fascicle 10).Armed Forces Institute of Pathology Washington D.C. 1994

5. T-cell co-stimulation by the CD28 ligand B7 is involved in the immune response leading to rejection of a spontaneously regressive tumor

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