Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenon

Author:

Bernard Tanya,Gale Rosemary E.,Evans Jane P. M.,Linch David C.

Publisher

Wiley

Subject

Hematology

Reference34 articles.

1. Analysis of granulocyte colony stimulating factor receptor isoforms, polymorphisms and mutations in normal haemopoietic cells and acute myeloid leukaemia blasts;Bernard;British Journal of Haematology,1996

2. Assessment of G-CSF and GM-CSF mRNA expression in peripheral blood mononuclear cells from patients with severe congenital neutropenia and in human myeloid cell lines;Bernhardt;Experimental Hematology,1993

3. Effects of recombinant human granulocyte colony-stimulating factor on neutropenia in patients with congenital agranulocytosis;Bonilla;New England Journal of Medicine,1989

4. Long-term safety of treatment with recombinant human granulocyte colony-stimulating factor (r-metHuG-CSF) in patients with severe congenital neutropenias;Bonilla;British Journal of Haematology,1994

5. Nonrandom X-inactivation patterns in normal females: Lyonization ratios vary with age;Busque;Blood,1996

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