Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenon
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.1998.00652.x/fullpdf
Reference34 articles.
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2. Assessment of G-CSF and GM-CSF mRNA expression in peripheral blood mononuclear cells from patients with severe congenital neutropenia and in human myeloid cell lines;Bernhardt;Experimental Hematology,1993
3. Effects of recombinant human granulocyte colony-stimulating factor on neutropenia in patients with congenital agranulocytosis;Bonilla;New England Journal of Medicine,1989
4. Long-term safety of treatment with recombinant human granulocyte colony-stimulating factor (r-metHuG-CSF) in patients with severe congenital neutropenias;Bonilla;British Journal of Haematology,1994
5. Nonrandom X-inactivation patterns in normal females: Lyonization ratios vary with age;Busque;Blood,1996
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1. Ultra-Sensitive CSF3R Deep Sequencing in Patients With Severe Congenital Neutropenia;Frontiers in Immunology;2019-02-28
2. Mechanisms of leukemic transformation in congenital neutropenia;Current Opinion in Hematology;2019-01
3. Chronic neutrophilic leukemia;Current Opinion in Hematology;2015-03
4. Granulocyte Colony-Stimulating Factor Receptor Mutations in Myeloid Malignancy;Frontiers in Oncology;2014-05-01
5. Severe congenital neutropenia, a genetically heterogeneous disease group with an increased risk of AML/MDS;Pediatric Reports;2011-06-22
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