Differences in circular muscle contraction and peristaltic motor inhibition caused by tachykinin NK1 receptor agonists in the guinea-pig small intestine
Author:
Publisher
Wiley
Subject
Gastroenterology,Endocrine and Autonomic Systems,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2982.2000.00199.x/fullpdf
Reference42 articles.
1. Evidence for the involvement of substance P in the atropine-resistant peristalsis of the guinea-pig ileum
2. Substance P enteric neurons mediate non-colinergic transmission to the circular muscle of the guinea-pig intestine
3. Tachykinin NK1 and NK2 receptor-mediated control of peristaltic propulsion in the guinea-pig small intestine in vitro
4. Roles of neuronal NK1 and NK3 receptors in synaptic transmission during motility reflexes in the guinea-pig ileum
5. Tachykinins in the gut. Part I. Expression, release and motor function
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3. Peristalsis and fecal pellet propulsion do not require nicotinic, purinergic, 5-HT3, or NK3 receptors in isolated guinea pig distal colon;American Journal of Physiology-Gastrointestinal and Liver Physiology;2010-06
4. A randomized, placebo-controlled, crossover, double-blind trial of the NK1 receptor antagonist aprepitant on gastrointestinal motor function in healthy humans;Alimentary Pharmacology & Therapeutics;2008-01-18
5. Substance P Methyl Ester;xPharm: The Comprehensive Pharmacology Reference;2007
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