The acute inflammatory response in CNS following injection of prion brain homogenate or normal brain homogenate-Reference-Cited by-同舟云学术

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The acute inflammatory response in CNS following injection of prion brain homogenate or normal brain homogenate

Published:1999-01 Issue:1 Volume:25 Page:19-27
ISSN:0305-1846
Container-title:Neuropathology and Applied Neurobiology
language:en
Short-container-title:Neuropathology and Applied Neurobiology

Author:

Betmouni ¹,Perry ¹

Affiliation:

1. CNS Inflammation Group, Department of Pharmacology, Oxford, UK

Publisher

Wiley

Subject

Physiology (medical),Neurology (clinical),Neurology,Histology,Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1046/j.1365-2990.1999.00153.x

Reference37 articles.

1. Intracerebral injection of proinflammatory cytokines or leukocyte chemotaxins induces minimal myelomonocytic cell recruitment to the parenchyma of the central nervous system.

2. The kinetics and morphological characteristics of the macrophage-microglial response to kainic acid-induced neuronal degeneration

3. Microglia is a component of the prion protein amyloid plaque in the Gerstmann-Str�ussler-Scheinker syndrome

4. Prion protein immunocytochemistry – UK five centre consensus report.;Bell JE;Neuropathol Appl Neurobiol,1997

5. EVIDENCE FOR AN EARLY INFLAMMATORY RESPONSE IN THE CENTRAL NERVOUS SYSTEM OF MICE WITH SCRAPIE

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1. Prion potentiation after life-long dormancy in mice devoid of PrP;Brain Communications;2021-04-01

2. Let’s make microglia great again in neurodegenerative disorders;Journal of Neural Transmission;2017-10-12

3. The ME7 prion model of neurodegeneration as a tool to understand and target neuroinflammation in Alzheimer’s disease;Drug Discovery Today: Disease Models;2017

4. Early Generation of New PrPSc on Blood Vessels after Brain Microinjection of Scrapie in Mice;mBio;2015-10-30

5. Early Hippocampal Synaptic Loss Precedes Neuronal Loss and Associates with Early Behavioural Deficits in Three Distinct Strains of Prion Disease;PLoS ONE;2013-06-26

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