Cardiac hypertrophy and failure: lessons learned from genetically engineered mice
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-201X.2001.00890.x/fullpdf
Reference78 articles.
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2. MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failure;Arber;Cell,1997
3. Beta-adrenergic receptor blockade arrests myocyte damage and preserves cardiac function in the transgenic Gsα mouse;Asai;J Clin Invest,1999
4. Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse hearts;Baker;Circ Res,1998
5. Increased protein kinase C activity and expression of Ca2+-sensitive isoforms in the failing human heart;Bowling;Circulation,1999
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