Absence of p53 gene mutations in skin fibroblasts derived from patients with systemic sclerosis
Author:
Publisher
Wiley
Subject
Clinical Biochemistry,Biochemistry,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2362.2002.00943.x/fullpdf
Reference10 articles.
1. p53, the cellular gatekeeper for growth and division;Levine;Cell,1997
2. Analysis of p53 gene mutations in keloids using polymerase chain reaction-based single-strand conformational polymorphism and DNA sequencing;Saed;Arch Dermatol,1998
3. Dominant-negative p53 mutations in rheumatoid arthritis;Han;Arthritis Rheum,1999
4. Lymphoma development in Sjogren’s syndrome: novel p53 mutations;Tapinos;Arthritis Rheum,1999
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Expressions of p53 and PUMA in fibroblasts of systemic sclerosis patients are normal at transcription level;Journal of Cosmetic Dermatology;2017-09-14
2. Increased expression of p53 and p21 (Waf1/Cip1) in the lesional skin of bleomycin-induced scleroderma;Archives of Dermatological Research;2005-04-01
3. Over-expression of TATA binding protein (TBP) and p53 and autoantibodies to these antigens are features of systemic sclerosis, systemic lupus erythematosus and overlap syndromes;Clinical and Experimental Immunology;2004-05-04
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