p53 intronic point mutation, aberrant splicing and telomeric associations in a case of B-chronic lymphocytic leukaemia
Author:
Publisher
Wiley
Subject
Hematology
Reference31 articles.
1. Hemi‐ or homozygosity: a requirement for some but not other p53 mutant proteins to accumulate and exert a pathogenetic effect
2. p53 Expression in B-Cell Chronic Lymphocytic Leukemia: A Marker of Disease Progression and Poor Prognosis
3. Oligoclonal B-cell leukemia characterized by spontaneous cell division and telomere association
4. p53 gene deletion predicts for poor survival and non-response to therapy with purine analogs in chronic B-cell leukemias
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1. Overexpression and Mutation of p53 Exons 4–8 in Canine Intestinal Adenocarcinoma;Journal of Comparative Pathology;2020-02
2. Chronic lymphocytic leukemia with isochromosome 17q: An aggressive subgroup associated with TP53 mutations and complex karyotypes;Cancer Letters;2017-11
3. Immunohistochemical correlates of TP53 somatic mutations in cancer;Oncotarget;2016-09-08
4. Mutation of p53 Gene and Its Correlation with the Clinical Outcome in Dogs with Lymphoma;Journal of Veterinary Internal Medicine;2015-12-17
5. Alternative Splicing in Chronic Myeloid Leukemia (CML): A Novel Therapeutic Target?;Current Cancer Drug Targets;2013-08-31
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