A case of purpura fulminans is caused by homozygous Δ8857 mutation (protein C-Nagoya) and successfully treated with activated protein C concentrate
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2000.02230.x/fullpdf
Reference12 articles.
1. Inherited protein C deficiency and coumarin-responsive chronic relapsing purpura fulminans in a newborn infant;Branson;Lancet,1983
2. Protein-C: biochemistry, physiology, and clinical implications;Esmon;Blood,1983
3. Characterization of a cDNA coding for human protein C;Foster;Proceedings of the National Academy of Sciences of the United States of America,1984
4. Deficiency of protein C in congenital thrombotic disease;Griffin;Journal of Clinical Investigation,1981
5. Direct detection of activated protein C in blood from human subjects;Gruber;Blood,1992
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