IMPLICATIONS OF RECENT INSIGHTS INTO THE PATHOPHYSIOLOGY OF PAROXYSMAL NOCTURNAL HAEMOGLOBINURIA

Author:

Hillmen Peter,Richards Stephen J.

Publisher

Wiley

Subject

Hematology

Reference56 articles.

1. Clonal populations of hematopoietic cells with paroxysmal nocturnal hemoglobinuria genotype and phenotype are present in normal individuals;Araten;Proceedings of the National Academy of Sciences of the USA,1999

2. Affected paroxysmal nocturnal hemoglobinuria T lymphocytes harbour a common defect in assembly of N-acetyl-D-glucosamine inositol phospholipid corresponding to that in class A Thy-1- murine lymphoma mutants;Armstrong;Journal of Biological Chemistry,1992

3. Biochemical studies on leucocytes. II. Phosphatase activity in chronic lymphatic leucemia, acute leucemia and miscellaneous hematologic conditions;Beck;Journal of Laboratory Clinical Medicine,1951

4. Paroxysmal nocturnal haemoglobinuria (PNH) is caused by somatic mutations in the PIG-A gene;Bessler;EMBO Journal,1994a

5. Somatic mutations and cellular selection in paroxysmal nocturnal haemoglobinuria;Bessler;Lancet,1994b

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