recessive spotting: a linked locus that interacts withW/Kitbut is not allelic

Author:

Bennett Dorothy C.1,Trayner Ian D.1,Piao Xianhua2,Easty David J1,Klüppel Michael2,Alexander Warren S.3,Wagner Erwin F.3,Bernstein Alan2

Affiliation:

1. St. George's Hospital Medical School, Cranmer Terrace, London SW17 0RE, UK,

2. Samuel Lunenfield Research Institute, Mount Sinai Hospital, 600 University Avenue, Toronto, Canada M5G 1X5,

3. Research Institute for Molecular Pathology, Dr Bohr‐Gasse 7, A‐1030 Vienna, Austria

Publisher

Wiley

Subject

Cell Biology,Genetics

Reference43 articles.

1. Expression of functional c-kit receptors rescues the genetic defect of W mutant mast cells.

2. Genetics, Development, and Malignancy of Melanocytes

3. A line of non-tumorigenic mouse melanocytes, syngeneic with the B16 melanoma and requiring a tumour promoter for growth

4. 4R. Blouin, and A. Bernstein (1993 ) TheWhite spottingandSteelhereditary anemias in the mouse. In:Clinical disorders and and experimental models of erythropoietic failure(eds S.A. Feig & M.H. Freedman), pp. 157, 173 . Ann. Arbor, Michigan: CRC Press.

5. A 1.8-Mb YAC contig spanning three members of the receptor tyrosine kinase gene family (Pdgfra, Kit, and Flk1) on mouse chromosome 5

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