Phosphatidylinositol-specific-phospholipase C cleaves urokinase plasminogen activator receptor from the cell surface and leads to inhibition of pemphigus-IgG-induced acantholysis in DJM-1 cells, a squamous cell carcinoma line
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2230.2001.00818.x/fullpdf
Reference25 articles.
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2. The pathogenic role of pemphigus antibodies and proteinase in epidermal acantholysis;Morioka;J Invest Dermatol,1981
3. Anti-cell surface pemphigus antibody stimulates plasminogen activator activity of human epidermal cells. A mechanism for the loss of epidermal adhesion and blister formation;Hashimoto;J Exp Med,1983
4. Targeted disruption of the pemphigus vulgaris antigen (desmoglein 3) gene in mice causes loss of keratinocyte cell adhesion with a phenotype similar to pemphigus vulgaris;Koch;J Cell Biol,1997
5. A pathogenic autoantibody, pemphigus vulgaris IgG, induces phosphorylation of desmoglein 3, and its dissociation from plakoglobin in cultured keratinocytes;Aoyama;Eur J Immunol,1999
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