The role of heterocellular hereditary persistence of fetal haemoglobin in β0-thalassaemia intermedia
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2001.03042.x/fullpdf
Reference31 articles.
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2. Genetic interactions in thalassemia intermedia: analysis of β-mutations, α-genotype, γ-promoters, and β-LCR hypersensitive site 2 and 4 in Italian patients;Camaschella;American Journal of Hematology,1995
3. Interaction between homozygous β0-thalassaemia and the Swiss type of hereditary persistence of fetal haemoglobin;Cappellini;British Journal of Haematology,1981
4. A β-thalassemia lesion abolishes the same MstII sites as the sickle mutation;Chang;Nucleic Acids Research,1983
5. Dissecting the loci controlling fetal hemoglobin production on chromosomes 11p and 6q by the regressive approach;Craig;Nature Genetics,1996
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