Resolution of Holliday junctions by RuvABC prevents dimer formation in rep mutants and UV-irradiated cells
Author:
Publisher
Wiley
Subject
Molecular Biology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2958.2000.01989.x/fullpdf
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1. sbcB sbcC null mutations allow RecF-mediated repair of arrested replication forks in rep recBC mutants
2. Two related recombinases are required for site-specific recombination at dif and cer in E. coli K12
3. Plasmid pSC101 harbors a recombination site, psi, which is able to resolve plasmid multimers and to substitute for the analogous chromosomal Escherichia coli site dif
4. A broadening view of recombinational DNA repair in bacteria
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