Acquired junctional epidermolysis bullosa associated with IgG autoantibodies to the beta subunit of laminin-5
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2133.1998.02038.x/fullpdf
Reference29 articles.
1. A passive transfer model of the organ-specific autoimmune disease, bullous pemphigoid, using antibodies generated against the hemidesmosomal antigen, BP180.
2. Passive transfer of autoantibodies from a patient with mutilating epidermolysis bullosa acquisita induces specific alterations in the skin of neonatal mice
3. Passive transfer of anti-laminin 5 antibodies induces subepidermal blisters in neonatal mice.
4. Cicatricial Pemphigoid Autoantibodies React with Multiple Sites on the BP180 Extracellular Domain
5. Anti-Basement Membrane Autoantibodies in Patients with Anti-Epiligrin Cicatricial Pemphigoid Bind the α Subunit of Laminin 5
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1. Pemphigoid ohne Schleimhautbeteiligung, mit IgG‐Antikörpern gegen die β3‐Untereinheit von Laminin‐332;JDDG: Journal der Deutschen Dermatologischen Gesellschaft;2024-04
2. Pemphigoid without mucous involvement showing IgG antibodies to the β3 subunit of laminin‐332;JDDG: Journal der Deutschen Dermatologischen Gesellschaft;2024-02-07
3. Spontaneous autoimmune subepidermal blistering diseases in animals: a comprehensive review;BMC Veterinary Research;2023-02-27
4. Ocular involvement in epidermolysis bullosa acquisita with long-term follow-up;British Journal of Ophthalmology;2019-05-14
5. Acquired Subepidermal Bullous Diseases;Evidence-Based Dermatology;2014-06-06
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