Erythrocyte Na+ ,K+ -ATPase and nasal potential in pseudohypoaldosteronism*
Author:
Publisher
Wiley
Subject
Endocrinology, Diabetes and Metabolism,Endocrinology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2265.2002.01503.x/fullpdf
Reference35 articles.
1. Aldosterone receptor deficiency in pseudohypoaldosteronism;Armanini;New England Journal of Medicine,1985
2. Reduced Na+,K+-ATPase activity in patients with pseudohypoaldosteronism;Bistritzer;Pediatric Research,1994
3. Severe pseudohypoaldosteronism in a pair of twins not associated with hydramnion;Bistritzer;Pediatric Nephrology,1996
4. Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1;Chang;Nature Genetics,1996
5. A salt-wasting syndrome in infancy;Cheek;Archives of Disease in Childhood,1958
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Syndromes of impaired ion handling in the distal nephron: pseudohypoaldosteronism and familial hyperkalemic hypertension;Hormones;2012-01
2. A novel splice site mutation of the beta subunit gene of epithelial sodium channel (ENaC) in one Turkish patient with a systemic form of pseudohypoaldosteronism type 1;Journal of Pediatric Endocrinology and Metabolism;2012-01-01
3. Renin–aldosterone response, urinary Na/K ratio and growth in pseudohypoaldosteronism patients with mutations in epithelial sodium channel (ENaC) subunit genes;The Journal of Steroid Biochemistry and Molecular Biology;2008-09
4. Structure and Function of CLCA Proteins;Physiological Reviews;2005-07
5. Pseudohypoaldosteronism: kidney, lungs and colon*;Clinical Endocrinology;2002-05
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