Mutation in the ATP-binding site of BCR-ABL in a patient with chronic myeloid leukaemia with increasing resistance to STI571-Reference-Cited by-同舟云学术

Mutation in the ATP-binding site of BCR-ABL in a patient with chronic myeloid leukaemia with increasing resistance to STI571

Published:2002-10 Issue:1 Volume:119 Page:109-111
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:

Author:

Barthe Christophe,Gharbi Marie-josée,Lagarde Valérie,Chollet Claudine,Cony-Makhoul Pascale,Reiffers Josy,Goldman John M.,Melo Junia V.,Mahon François Xavier

Publisher

Wiley

Subject

Hematology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2002.03708.x/fullpdf

Reference10 articles.

1. BCR-ABL gene mutations in relation to clinical resistance of Philadelphia-positive leukaemia to STI571;Von Bubnoff;Lancet,2002

2. The tyrosine kinase inhibitor CGP57148B selectively inhibits the growth of BCR-ABL-positive cells;Deininger;Blood,1997

3. Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome;Druker;New England Journal of Medicine,2001a

4. Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinase in chronic myeloid leukemia;Druker;New England Journal of Medicine,2001b

5. Role of alpha1 acid glycoprotein in the in vivo resistance of human BCR-ABL (+) leukemic cells to the abl inhibitor STI571;Gambacorti-Passerini;Journal of the National Cancer Institute,2000

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