Epileptogenesis Causes Acute and Chronic Increases in GABAA Receptor Endocytosis That Contributes to the Induction and Maintenance of Seizures in the Hippocampal Culture Model of Acquired Epilepsy-Reference-Cited by-同舟云学术

Epileptogenesis Causes Acute and Chronic Increases in GABAA Receptor Endocytosis That Contributes to the Induction and Maintenance of Seizures in the Hippocampal Culture Model of Acquired Epilepsy

Published:2004-04-14 Issue:3 Volume:310 Page:871-880
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Blair Robert E.,Sombati Sompong,Lawrence David C.,McCay Brendan D.,DeLorenzo Robert J.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference36 articles.

1. Molecular biology of the GABAA receptor: the receptor/channel superfamily

2. Assembly and intracellular trafficking of GABAA receptors

3. Long-lasting decrease in neuronal Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent seizures

4. Selective changes in single cell GABAA receptor subunit expression and function in temporal lobe epilepsy

5. Role of ampa receptor endocytosis in synaptic plasticity

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