Infection-Induced Kinin B1Receptors in Human Pulmonary Fibroblasts: Role of Intact Pathogens and p38 Mitogen-Activated Protein Kinase-Dependent Signaling-Reference-Cited by-同舟云学术

Infection-Induced Kinin B1Receptors in Human Pulmonary Fibroblasts: Role of Intact Pathogens and p38 Mitogen-Activated Protein Kinase-Dependent Signaling

Published:2005-03-02 Issue:3 Volume:313 Page:1231-1238
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Phagoo Stephen B.,Reddi Krisanavane,Silvallana Bertrand J.,Leeb-Lundberg L. M. Fredrik,Warburton David

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference40 articles.

1. Kinin B1receptors: key G-protein-coupled receptors and their role in inflammatory and painful processes

2. Treatment of Severe Systemic Inflammatory Response Syndrome and Sepsis With a Novel Bradykinin Antagonist, Deltibant (CP-0127)Results of a Randomized, Double-blind, Placebo-Controlled Trial

3. Differential modulation of murine lung inflammation by bradykinin B1 and B2 selective receptor antagonists

4. p38 stress-activated protein kinase inhibitor reverses bradykinin B1 receptor-mediated component of inflammatory hyperalgesia

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