Safinamide Modulates Striatal Glutamatergic Signaling in a Rat Model of Levodopa-Induced Dyskinesia-Reference-Cited by-同舟云学术

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Safinamide Modulates Striatal Glutamatergic Signaling in a Rat Model of Levodopa-Induced Dyskinesia

Published:2018-10-05 Issue:3 Volume:367 Page:442-451
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Gardoni F.,Morari M.,Kulisevsky J.,Brugnoli A.,Novello S.,Pisanò C. A.,Caccia C.,Mellone M.,Melloni E.,Padoani G.,Sosti V.,Vailati S.,Keywood C.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference54 articles.

1. Increased D1dopamine receptor signaling in levodopa-induced dyskinesia

2. Postsynaptic density protein 95-regulated NR2B tyrosine phosphorylation and interactions of Fyn with NR2B in levodopa-induced dyskinesia rat models;Ba;Drug Des Devel Ther,2014

3. Rebalance of Striatal NMDA/AMPA Receptor Ratio Underlies the Reduced Emergence of Dyskinesia During D2-Like Dopamine Agonist Treatment in Experimental Parkinson's Disease

4. Identification of the Ca2+/Calmodulin-dependent Protein Kinase II Regulatory Phosphorylation Site in the α-Amino-3-hydroxyl-5-methyl4-isoxazole-propionate-type Glutamate Receptor

5. Pathophysiology of L-dopa-induced motor and non-motor complications in Parkinson's disease

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1. Functional efficacy of the MAO-B inhibitor safinamide in murine substantia nigrapars compactadopaminergic neuronsin vitro: a comparative study with tranylcypromine;2024-06-01

2. Suppression of presynaptic corticostriatal glutamate activity attenuates L-dopa-induced dyskinesia in 6-OHDA-lesioned Parkinson's disease mice;Neurobiology of Disease;2024-04

3. Application of a Quality by Design Approach to Develop a Simple, Fast, and Sensitive UPLC-MS/MS Method for Quantification of Safinamide, an Antiparkinson’s Drug, in Plasma;Separations;2023-08-28

4. Future Directions for Developing Non-dopaminergic Strategies for the Treatment of Parkinson’s Disease;Current Neuropharmacology;2023-07-31

5. Monoamine-oxidase Type B Inhibitors and Cognitive Functions in Parkinson’s Disease: Beyond the Primary Mechanism of Action;Current Neuropharmacology;2023-05

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