Agonist Activation of δ-Opioid Receptor but not μ-Opioid Receptor Potentiates Fetal Calf Serum or Tyrosine Kinase Receptor-Mediated Cell Proliferation in a CellLine-Specific Manner-Reference-Cited by-同舟云学术

Agonist Activation of δ-Opioid Receptor but not μ-Opioid Receptor Potentiates Fetal Calf Serum or Tyrosine Kinase Receptor-Mediated Cell Proliferation in a CellLine-Specific Manner

Published:1997-01-01 Issue:1 Volume:51 Page:152-160
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Law P. Y.,McGinn T. M.,Campbell K. M.,Erickson L. E.,Loh H.H.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference43 articles.

1. K-Opioid Agonist Modulation of [3H]Thymidine Incorporation into DNA: Evidence for the Involvement of Pertussis Toxin-Sensitive G Protein-Coupled Phosphoinositide Turnover

2. Effects of opiates on the growth of neuron-enriched cultures from chick embryonic brain

3. Opioid-dependent growth of glial cultures: Suppression of astrocyte DNA synthesis by met-enkephalin

4. Endogenous opioid systems regulate growth of neural tumor cells in culture

5. Opioid antagonist (naltrexone) stimulation of cell proliferation in human and animal neuroblastoma and human fibrosarcoma cells in culture

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