2,3,7,8-Tetrachlorodibenzo-p-dioxin Suppresses Tumor Necrosis Factor-α and Anti-CD40–Induced Activation of NF-κB/Rel in Dendritic Cells: p50 Homodimer Activation Is Not Affected-Reference-Cited by-同舟云学术

2,3,7,8-Tetrachlorodibenzo-p-dioxin Suppresses Tumor Necrosis Factor-α and Anti-CD40–Induced Activation of NF-κB/Rel in Dendritic Cells: p50 Homodimer Activation Is Not Affected

Published:2002-09-01 Issue:3 Volume:62 Page:722-728
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Ruby Carl E.,Leid Mark,Kerkvliet Nancy I.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference38 articles.

1. Immunosuppression by Glucocorticoids: Inhibition of NF-κB Activity Through Induction of IκB Synthesis

2. THE NF-κB AND IκB PROTEINS: New Discoveries and Insights

3. Dendritic cells and the control of immunity

4. NF-κB RelA-deficient Lymphocytes: Normal Development of T Cells and B Cells, Impaired Production of IgA and IgG1 and Reduced Proliferative Responses

5. Isolation of intact nuclei for nuclear extract preparation from a fragile B-lymphocyte cell line.;Dyer;Biotechniques,1995

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