Fluorizoline Blocks the Interaction between Prohibitin-2 and γ-Glutamylcyclotransferase and Induces p21Waf1/Cip1 Expression in MCF7 Breast Cancer Cells-Reference-Cited by-同舟云学术

Fluorizoline Blocks the Interaction between Prohibitin-2 and γ-Glutamylcyclotransferase and Induces p21Waf1/Cip1 Expression in MCF7 Breast Cancer Cells

Published:2021-12-03 Issue:2 Volume:101 Page:78-86
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Takagi Hiroko,Moyama Chiami,Taniguchi Keiko,Ando Kota,Matsuda Ryohei,Ando Shota,Ii Hiromi,Kageyama Susumu,Kawauchi Akihiro,Chouha Nora,Désaubry Laurent,Nakata Susumu

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference21 articles.

1. Prohibitin: a potential therapeutic target in tyrosine kinase signaling;Ande;Signal Transduct Target Ther,2017

2. PHB in Cardiovascular and Other Diseases: Present Knowledge and Implications

3. The prohibitin-binding compound fluorizoline induces apoptosis in chronic lymphocytic leukemia cells through the upregulation of NOXA and synergizes with ibrutinib, 5-aminoimidazole-4-carboxamide riboside or venetoclax

4. WAF1, a potential mediator of p53 tumor suppression

5. The prohibitin-binding compound fluorizoline affects multiple components of the translational machinery and inhibits protein synthesis

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