RhoA/Phosphatidylinositol 3-Kinase/Protein Kinase B/Mitogen-Activated Protein Kinase Signaling after Growth Arrest–Specific Protein 6/Mer Receptor Tyrosine Kinase Engagement Promotes Epithelial Cell Growth and Wound Repair via Upregulation of Hepatocyte Growth Factor in Macrophages-Reference-Cited by-同舟云学术

RhoA/Phosphatidylinositol 3-Kinase/Protein Kinase B/Mitogen-Activated Protein Kinase Signaling after Growth Arrest–Specific Protein 6/Mer Receptor Tyrosine Kinase Engagement Promotes Epithelial Cell Growth and Wound Repair via Upregulation of Hepatocyte Growth Factor in Macrophages

Published:2014-06-17 Issue:3 Volume:350 Page:563-577
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Lee Ye-Ji,Park Hyun-Jung,Woo So-Youn,Park Eun-Mi,Kang Jihee Lee

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference54 articles.

1. Neutrophil Defensins Enhance Lung Epithelial Wound Closure and Mucin Gene ExpressionIn Vitro

2. TNF-α, IL-6, and IL-1 expression is inhibited by GAS6 in monocytes/macrophages

3. Novel Mechanism for Gonadotropin-Releasing Hormone Neuronal Migration Involving Gas6/Ark Signaling to p38 Mitogen-Activated Protein Kinase

4. Animal models of wound repair: Are they cutting it?

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