Mechanisms Underlying Benign and Reversible Unconjugated Hyperbilirubinemia Observed with Faldaprevir Administration in Hepatitis C Virus Patients-Reference-Cited by-同舟云学术

Mechanisms Underlying Benign and Reversible Unconjugated Hyperbilirubinemia Observed with Faldaprevir Administration in Hepatitis C Virus Patients

Published:2014-09-09 Issue:2 Volume:351 Page:403-412
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Sane Rucha S.,Steinmann Gerhard G.,Huang Qihong,Li Yongmei,Podila Lalitha,Mease Kirsten,Olson Stephen,Taub Mitchell E.,Stern Jerry O.,Nehmiz Gerhard,Böcher Wulf O.,Asselah Tarik,Tweedie Donald

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference32 articles.

1. Drug-induced Hyperbilirubinemia and the Clinical Influencing Factors

2. Interaction of HIV protease inhibitors with OATP1B1, 1B3, and 2B1

3. Second-wave IFN-based triple therapy for HCV genotype 1 infection: simeprevir, faldaprevir and sofosbuvir

4. Inhibition of human organic anion transporting polypeptide OATP 1B1 as a mechanism of drug-induced hyperbilirubinemia

5. Evaluating the In Vitro Inhibition of UGT1A1, OATP1B1, OATP1B3, MRP2, and BSEP in Predicting Drug-Induced Hyperbilirubinemia

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