Dual Regulation of Tumor Necrosis Factor-α-Induced CCL2/Monocyte Chemoattractant Protein-1 Expression in Vascular Smooth Muscle Cells by Nuclear Factor-κB and Activator Protein-1: Modulation by Type III Phosphodiesterase Inhibition-Reference-Cited by-同舟云学术

Dual Regulation of Tumor Necrosis Factor-α-Induced CCL2/Monocyte Chemoattractant Protein-1 Expression in Vascular Smooth Muscle Cells by Nuclear Factor-κB and Activator Protein-1: Modulation by Type III Phosphodiesterase Inhibition

Published:2004-02-20 Issue:3 Volume:309 Page:978-986
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Chen Yung-Ming,Chiang Wen-Chih,Lin Shuei-Liong,Wu Kwan-Dun,Tsai Tun-Jun,Hsieh Bor-Shen

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference44 articles.

1. Role of Phosphodiesterase 3 in NO/cGMP-Mediated Antiinflammatory Effects in Vascular Smooth Muscle Cells

2. Critical Role of c-Jun N-Terminal Protein Kinase Activation in Troglitazone-Induced Apoptosis of Human HepG2 Hepatoma Cells

3. Nuclear Factor-κB — A Pivotal Transcription Factor in Chronic Inflammatory Diseases

4. Signal transduction by tumor necrosis factor and its relatives

5. SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase

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