cAMP and Extracellular Signal-Regulated Kinase Signaling in Response to d-Amphetamine and Methylphenidate in the Prefrontal Cortex in Vivo: Role of β1-Adrenoceptors-Reference-Cited by-同舟云学术

cAMP and Extracellular Signal-Regulated Kinase Signaling in Response to d-Amphetamine and Methylphenidate in the Prefrontal Cortex in Vivo: Role of β1-Adrenoceptors

Published:2005-05-12 Issue:2 Volume:68 Page:421-429
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Pascoli Vincent,Valjent Emmanuel,Corbillé Anne-Gaëlle,Corvol Jean-Christophe,Tassin Jean-Pol,Girault Jean-Antoine,Hervé Denis

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference38 articles.

1. The Contribution of α2-Noradrenergic Mechanisms to Prefrontal Cortical Cognitive Function

2. 5-HT2A and α1b-adrenergic receptors entirely mediate dopamine release, locomotor response and behavioural sensitization to opiates and psychostimulants

3. Control of GluR1 AMPA Receptor Function by cAMP-Dependent Protein Kinase

4. Direct and indirect interactions between cannabinoid CB1 receptor and group II metabotropic glutamate receptor signalling in layer V pyramidal neurons from the rat prefrontal cortex

5. Relationship between low-dose amphetamine-induced arousal and extracellular norepinephrine and dopamine levels within prefrontal cortex

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