Yet Another Function of p53—The Switch That Determines Whether Radiation-Induced Autophagy Will Be Cytoprotective or Nonprotective: Implications for Autophagy Inhibition as a Therapeutic Strategy-Reference-Cited by-同舟云学术

Yet Another Function of p53—The Switch That Determines Whether Radiation-Induced Autophagy Will Be Cytoprotective or Nonprotective: Implications for Autophagy Inhibition as a Therapeutic Strategy

Published:2015-02-09 Issue:5 Volume:87 Page:803-814
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Chakradeo Shweta,Sharma Khushboo,Alhaddad Aisha,Bakhshwin Duaa,Le Ngoc,Harada Hisashi,Nakajima Wataru,Yeudall W. Andrew,Torti Suzy V.,Torti Frank M.,Gewirtz David A.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference42 articles.

1. Autophagy delays apoptotic death in breast cancer cells following DNA damage

2. Autophagy inhibition enhances therapy-induced apoptosis in a Myc-induced model of lymphoma

3. Blocked Autophagy Sensitizes Resistant Carcinoma Cells to Radiation Therapy

4. Chapter 12 Monitoring Autophagic Degradation of p62/SQSTM1

5. Inhibition of Macroautophagy Triggers Apoptosis

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