Differential Contribution of Subunit Interfaces to α9α10 Nicotinic Acetylcholine Receptor Function-Reference-Cited by-同舟云学术

Differential Contribution of Subunit Interfaces to α9α10 Nicotinic Acetylcholine Receptor Function

Published:2017-01-09 Issue:3 Volume:91 Page:250-262
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Boffi Juan Carlos,Marcovich Irina,Gill-Thind JasKiran K.,Corradi Jeremías,Collins Toby,Lipovsek María Marcela,Moglie Marcelo,Plazas Paola V.,Craig Patricio O.,Millar Neil S.,Bouzat Cecilia,Elgoyhen Ana Belén

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference71 articles.

1. Contributions of the non‐α subunit residues (loop D) to agonist binding and channel gating in the muscle nicotinic acetylcholine receptor

2. Stoichiometry for activation of neuronal 7 nicotinic receptors

3. Topology of ligand binding sites on the nicotinic acetylcholine receptor

4. The SWISS-MODEL workspace: a web-based environment for protein structure homology modelling

5. Molecular basis for the differential sensitivity of rat and human α9α10 nAChRs to α-conotoxin RgIA

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