Desformylflustrabromine Modulates α4β2 Neuronal Nicotinic Acetylcholine Receptor High- and Low-Sensitivity Isoforms at Allosteric Clefts Containing the β2 Subunit-Reference-Cited by-同舟云学术

Desformylflustrabromine Modulates α4β2 Neuronal Nicotinic Acetylcholine Receptor High- and Low-Sensitivity Isoforms at Allosteric Clefts Containing the β2 Subunit

Published:2015-05-29 Issue:2 Volume:354 Page:184-194
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Weltzin Maegan M.,Schulte Marvin K.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

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1. Evidence for a role of nicotinic acetylcholine receptors in schizophrenia

2. Two Tyrosine Residues on the α Subunit Are Crucial for Benzodiazepine Binding and Allosteric Modulation of γ-Aminobutyric AcidA Receptors

3. Comparative Alterations of Nicotinic and Muscarinic Binding Sites in Alzheimer's and Parkinson's Diseases

4. Calcium buffering and slow recovery kinetics of calcium-dependent outward current in molluscan neurones.

5. Pharmacology of recombinant γ-aminobutyric acidAreceptors rendered diazepam-insensitive by point-mutated α-subunits

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