Notoginsenoside R1 Protects against Neonatal Cerebral Hypoxic-Ischemic Injury through Estrogen Receptor-Dependent Activation of Endoplasmic Reticulum Stress Pathways-Reference-Cited by-同舟云学术

Notoginsenoside R1 Protects against Neonatal Cerebral Hypoxic-Ischemic Injury through Estrogen Receptor-Dependent Activation of Endoplasmic Reticulum Stress Pathways

Published:2016-02-18 Issue:3 Volume:357 Page:591-605
ISSN:1521-0103
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:Journal of Pharmacology and Experimental Therapeutics

Author:

Wang Y.,Tu L.,Li Y.,Chen D.,Wang S.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference54 articles.

1. ATF6 Is a Transcription Factor Specializing in the Regulation of Quality Control Proteins in the Endoplasmic Reticulum

2. Induction of ER stress in response to oxygen-glucose deprivation of cortical cultures involves the activation of the PERK and IRE-1 pathways and of caspase-12

3. Endoplasmic Reticulum Stress Interacts With Inflammation in Human Diseases

5. Endoplasmic reticulum Ca2+dysregulation and endoplasmic reticulum stress followingin vitroneuronal ischemia: role of Na+-K+-Cl-cotransporter

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