Role ofS-Nitrosation of Cysteine Residues in Long-Lasting Inhibitory Effect of Nitric Oxide on Arterial Tone

Author:

Alencar Jacicarlos L.,Lobysheva Irina,Geffard Michel,Sarr Mamadou,Schott Christa,Schini-Kerth Valérie B.,Nepveu Françoise,Stoclet Jean-Claude,Muller Bernard

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference47 articles.

1. Evidence for the formation of vasoactive nitric oxide stores by S-nitrosation of cysteine residues in isolated blood vessels exposed to S-nitrosoglutathione (Abstract).;Alencar;Br J Pharmacol,2002

2. Protein S-nitrosation as a mechanism of formation of releasable NO stores and long lasting decrease of arterial tone (Abstract).;Alencar;Nitric Oxide,2002

3. NO+, NO , and NO− Donation by S-Nitrosothiols: Implications for Regulation of Physiological Functions by S-Nitrosylation and Acceleration of Disulfide Formation

4. Diethyl Dithiocarbamate-Induced Decomposition ofS-Nitrosothiols

5. Indirect evidence for nitric oxide involvement in multiple sclerosis by characterization of circulating antibodies directed against conjugated S-nitrosocysteine

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