New Insights into Metabolic Signaling and Cell Survival: The Role of β-O-Linkage ofN-Acetylglucosamine
Author:
Publisher
American Society for Pharmacology & Experimental Therapeutics (ASPET)
Subject
Pharmacology,Molecular Medicine
Reference79 articles.
1. Mice lacking the poly(ADP-ribose) polymerase gene are resistant to pancreatic beta-cell destruction and diabetes development induced by streptozocin
2. Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associatedO-GlcNAc
3. Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2
4. HEXOSAMINE BIOSYNTHESIS AND PROTEIN O-GLYCOSYLATION
5. Alternative O-Glycosylation/O-Phosphorylation of the Murine Estrogen Receptor β
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1. An Overview of Glycosylation and its Impact on Cardiovascular Health and Disease;Frontiers in Molecular Biosciences;2021-11-16
2. Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarction;PLOS ONE;2020-11-30
3. Insights into the role of maladaptive hexosamine biosynthesis and O-GlcNAcylation in development of diabetic cardiac complications;Pharmacological Research;2017-02
4. O-linked β-N-acetylglucosamine modification of proteins is activated in post-ischemic brains of young but not aged mice: Implications for impaired functional recovery from ischemic stress;Journal of Cerebral Blood Flow & Metabolism;2015-09-30
5. High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation;Biochemical Journal;2015-03-20
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