Endothelium-Derived Nitric Oxide Inhibits the Relaxation of the Porcine Coronary Artery to Natriuretic Peptides by Desensitizing Big Conductance Calcium-Activated Potassium Channels of Vascular Smooth Muscle-Reference-Cited by-同舟云学术

Endothelium-Derived Nitric Oxide Inhibits the Relaxation of the Porcine Coronary Artery to Natriuretic Peptides by Desensitizing Big Conductance Calcium-Activated Potassium Channels of Vascular Smooth Muscle

Published:2010-03-23 Issue:1 Volume:334 Page:223-231
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Liang Chao Fan,Au Alice Lai Shan,Leung Susan Wai Sum,Ng Kwok Fu Jacobus,Félétou Michel,Kwan Yiu Wa,Man Ricky Ying Keung,Vanhoutte Paul M.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference40 articles.

1. Mechanisms of Action of Atrial Natriuretic Factor and C-Type Natriuretic Peptide

2. Gender and Relaxation to C-Type Natriuretic Peptide in Porcine Coronary Arteries

3. Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle

4. C-type natriuretic peptide: new candidate for endothelium-derived hyperpolarising factor

5. The BKCa Channel's Ca2+-binding Sites, Multiple Sites, Multiple Ions

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