Acute Inhibition of Ca2+/Calmodulin-Dependent Protein Kinase II Reverses Experimental Neuropathic Pain in Mice-Reference-Cited by-同舟云学术

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Acute Inhibition of Ca2+/Calmodulin-Dependent Protein Kinase II Reverses Experimental Neuropathic Pain in Mice

Published:2009-05-28 Issue:2 Volume:330 Page:650-659
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Chen Yan,Luo Fang,Yang Cheng,Kirkmire Chelsea M.,Wang Zaijie Jim

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference43 articles.

1. Colocalization of the μ-opioid receptor and calcium/calmodulin-dependent kinase II in distinct pain-processing brain regions

2. Localization of CaMKIIα in rat primary sensory neurons: increase in inflammation

3. Quantitative assessment of tactile allodynia in the rat paw

4. Involvement of phosphorylated Ca2+/calmodulin-dependent protein kinase II and phosphorylated extracellular signal-regulated protein in the mouse formalin pain model

5. Ca2+/calmodulin-dependent protein kinase II in the spinal cord contributes to neuropathic pain in a rat model of mononeuropathy

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