Protein Kinase C-Mediated Down-Regulation of β1-Adrenergic Receptor Gene Expression in Rat C6 Glioma Cells

Author:

Li Zhongwei,Vaidya Vidita A.,Alvaro John D.,Iredale Philip A.,Hsu Richard,Hoffman Ginger,Fitzgerald Laura,Curran Patricia K.,Machida Curtis A.,Fishman Peter H.,Duman Ronald S.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference40 articles.

1. Thyroid hormones transcriptionally regulate the β1-adrengergic receptor gene in cultured ventricular myocytes.;Bahouth;J Biol Chem,1991

2. Enhancement of adenylate cyclase activity in S49 lymphoma cells by phorbol esters.;Bell;J Biol Chem,1985

3. Regulation of adrenergic receptor function by phosphorylation. II. Effects of agonist occupancy on phosphorylation of α1- and β2-adrenergic receptors by protein kinase C and the cyclic AMP-dependent protein kinase.;Bouvier;J Biol Chem,1987

4. Phorbol-ester-induced phosphorylation of the β2-adrenergic receptor decreases its coupling to Gs

5. Protein kinase C α, δ, ϵ and ζ in C6glioma cells

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