Selective, Direct Activation of High-Conductance, Calcium-Activated Potassium Channels Causes Smooth Muscle Relaxation-Reference-Cited by-同舟云学术

Selective, Direct Activation of High-Conductance, Calcium-Activated Potassium Channels Causes Smooth Muscle Relaxation

Published:2012-01-12 Issue:4 Volume:81 Page:567-577
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Ponte Cristiano G.,McManus Owen B.,Schmalhofer William A.,Shen Dong-Ming,Dai Ge,Stevenson Andra,Sur Sylvie,Shah Tarak,Kiss Laszlo,Shu Min,Doherty James B.,Nargund Ravi,Kaczorowski Gregory J.,Suarez-Kurtz Guilherme,Garcia Maria L.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference38 articles.

1. Analgesic Effects of a Substituted N-Triazole Oxindole (TROX-1), a State-Dependent, Voltage-Gated Calcium Channel 2 Blocker

2. Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet

3. Vasoregulation by the β1 subunit of the calcium-activated potassium channel

4. BK channel β4 subunit reduces dentate gyrus excitability and protects against temporal lobe seizures

5. β-Adrenergic relaxation of mouse urinary bladder smooth muscle in the absence of large-conductance Ca2+-activated K+ channel

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