Valproic Acid Increases Formation of Reactive Oxygen Species and Induces Apoptosis in Postimplantation Embryos: A Role for Oxidative Stress in Valproic Acid-Induced Neural Tube Defects-Reference-Cited by-同舟云学术

Valproic Acid Increases Formation of Reactive Oxygen Species and Induces Apoptosis in Postimplantation Embryos: A Role for Oxidative Stress in Valproic Acid-Induced Neural Tube Defects

Published:2011-08-25 Issue:6 Volume:80 Page:979-987
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Tung Emily W. Y.,Winn Louise M.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference38 articles.

1. Embryoprotective Role of Endogenous Catalase in Acatalasemic and Human Catalase-Expressing Mouse Embryos Exposed in Culture to Developmental and Phenytoin-Enhanced Oxidative Stress

2. Changing trends in antiepileptic drug prescribing in girls of child-bearing potential

3. Vitamin E decreases valproic acid induced neural tube defects in mice

4. Stereoselective dysmorphogenicity of the enantiomers of the valproic acid analogue 2-N-propyl-4-pentynoic acid (4-yn-VPA): Cross-species evaluation in whole embryo culture

5. Abnormal development and increased 3-nitrotyrosine in copper-deficient mouse embryos

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