Abnormal Insulin-like Growth Factor 1 Signaling Regulates Neuropathic Pain by Mediating the Mechanistic Target of Rapamycin-Related Autophagy and Neuroinflammation in Mice-Reference-Cited by-同舟云学术

Abnormal Insulin-like Growth Factor 1 Signaling Regulates Neuropathic Pain by Mediating the Mechanistic Target of Rapamycin-Related Autophagy and Neuroinflammation in Mice

Published:2021-07-15 Issue:15 Volume:12 Page:2917-2928
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Chen Xin¹²,Le Yue²³,He Wan-you²,He Jian²,Wang Yun-hua²,Zhang Lei²,Xiong Qing-ming²,Zheng Xue-qin²,Liu Ke-xuan¹,Wang Han-bing²^ORCID

Affiliation:

1. Department of Anesthesiology, Nan Fang Hospital, Southern Medical University, Guangzhou 510515, Guangdong, China

2. Department of Anesthesiology, The First People’s Hospital of Foshan, 81# North of Ling Nan Road, Foshan 528000, Guangdong, China

3. Department of Anesthesiology, Renmin Hospital of Wuhan University, Wuhan 430060, Hubei, China

Funder

National Natural Science Foundation of China

GuangDong Basic and Applied Basic Research Foundation

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.1c00271

Reference44 articles.

1. Neuropathic pain

2. Neuropathic Pain: Principles of Diagnosis and Treatment

3. The role of Insulin-Like Growth Factor 1 (IGF-1) in brain development, maturation and neuroplasticity

4. Brain IGF-1 Receptors Control Mammalian Growth and Lifespan through a Neuroendocrine Mechanism

5. Antinociceptive action of intrathecally administered IGF-I and the expression of its receptor in rat spinal cord

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