Allosteric Activation of SIRT1 by 2,4-Dihydroxy-azaflavanone Averts MPP<sup>+</sup>-Mediated Dysfunction in Mitochondrial Biogenesis and Bioenergetics: Implications for Parkinson’s Disease-Reference-Cited by-同舟云学术

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Allosteric Activation of SIRT1 by 2,4-Dihydroxy-azaflavanone Averts MPP⁺-Mediated Dysfunction in Mitochondrial Biogenesis and Bioenergetics: Implications for Parkinson’s Disease

Published:2024-07-29 Issue:15 Volume:15 Page:2870-2883
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Pant Chitrakshi¹²,Sukumar Genji³⁴,Alli Vidya Jyothi⁵,Jadav Surender Singh²⁵^ORCID,Pabbaraja Srihari²³^ORCID,Kalivendi Shasi V.¹²^ORCID

Affiliation:

1. Department of Applied Biology, CSIR-Indian Institute of Chemical Technology, Uppal Road, Hyderabad 500007, India

2. Academy of Scientific and Innovative Research (AcSIR), Ghaziabad 201002, India

3. Department of Organic Synthesis and Process Chemistry, CSIR-Indian Institute of Chemical Technology, Uppal Road, Hyderabad 500007, India

4. Department of Chemistry, Adikavi Nannaya University, Rajamahendravaram, AP 533296, India

5. Department of Natural Products and Medicinal Chemistry, CSIR-Indian Institute of Chemical Technology, Uppal Road, Hyderabad 500007, India

Funder

Council of Scientific and Industrial Research, India

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.4c00244

Reference61 articles.

1. Biochemical aspects of Parkinson's disease

2. The role of parkin in familial and sporadic Parkinson's disease

3. Genetic etiology of Parkinson disease associated with mutations in the SNCA, PARK2, PINK1, PARK7, and LRRK2 genes: a mutation update

4. Mitochondrial dysfunction and mitophagy in Parkinson's: from familial to sporadic disease

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