Lipid-Chaperone Hypothesis: A Common Molecular Mechanism of Membrane Disruption by Intrinsically Disordered Proteins-Reference-Cited by-同舟云学术

Lipid-Chaperone Hypothesis: A Common Molecular Mechanism of Membrane Disruption by Intrinsically Disordered Proteins

Published:2020-12-03 Issue:24 Volume:11 Page:4336-4350
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Sciacca Michele F.¹,Lolicato Fabio²³^ORCID,Tempra Carmelo⁴⁵^ORCID,Scollo Federica⁵⁶,Sahoo Bikash R.⁷^ORCID,Watson Matthew D.⁸,García-Viñuales Sara¹,Milardi Danilo¹^ORCID,Raudino Antonio⁵,Lee Jennifer C.⁸^ORCID,Ramamoorthy Ayyalusamy⁷^ORCID,La Rosa Carmelo⁵^ORCID

Affiliation:

1. Istituto di Cristallografia, CNR, Catania 95126, Italy

2. Heidelberg University Biochemistry Center, Heidelberg 69120, Germany

3. Department of Physics, University of Helsinki, P.O. Box 64, Helsinki FI-00014, Finland

4. Institute of Organic Chemistry and Biochemistry, Prague 160 00, Czech Republic

5. Department of Chemical Sciences, University of Catania, Catania 95124, Italy

6. J. Heyrovský Institute of Physical Chemistry, Academy of Sciences of the Czech Republic, Prague 117 20, Czech Republic

7. Biophysics and Department of Chemistry, University of Michigan, Ann Arbor, Michigan 48109-1055, United States

8. National Institutes of Health, Bethesda, Maryland 20892-0001, United States

Funder

Universit? di Catania

National Heart, Lung, and Blood Institute

Grantov? Agentura Cesk? Republiky

Foundation for the National Institutes of Health

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.0c00588

Reference78 articles.

1. Protein Misfolding, Amyloid Formation, and Human Disease: A Summary of Progress Over the Last Decade

2. Protein Misfolding, Functional Amyloid, and Human Disease

3. Amyloidogenic Intrinsically Disordered Proteins: New Insights into Their Self-Assembly and Their Interaction with Membranes

4. The amyloid hypothesis of Alzheimer's disease at 25 years

5. The Interplay of Catalysis and Toxicity by Amyloid Intermediates on Lipid Bilayers: Insights from Type II Diabetes

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