Seeding Brain Protein Aggregation by SARS-CoV-2 as a Possible Long-Term Complication of COVID-19 Infection-Reference-Cited by-同舟云学术

Seeding Brain Protein Aggregation by SARS-CoV-2 as a Possible Long-Term Complication of COVID-19 Infection

Published:2020-11-04 Issue:22 Volume:11 Page:3704-3706
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Tavassoly Omid¹^ORCID,Safavi Farinaz²,Tavassoly Iman³

Affiliation:

1. Department of Chemistry, Simon Fraser University, Burnaby, British Columbia V5A 1S6, Canada

2. Neuroimmunology and Neurovirology Branch, National Institute of Neurological Disorders and Stroke, National Institute of Health, Bethesda, Maryland 20892, United States

3. C2i Genomics, 180 Varick Street, Sixth Floor, New York, New York 10014, United States

Funder

National Institute of Neurological Disorders and Stroke

W. Garfield Weston Foundation

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.0c00676

Reference8 articles.

1. Viruses and neurodegeneration

2. The viral protein corona directs viral pathogenesis and amyloid aggregation

3. Viral M45 and necroptosis‐associated proteins form heteromeric amyloid assemblies

4. NSs amyloid formation is associated with the virulence of Rift Valley fever virus in mice

5. A gut bacterial amyloid promotes α-synuclein aggregation and motor impairment in mice

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