Neuroinflammation-Modulating Agent SB1617 Enhances LC3-Associated Phagocytosis to Mitigate Tau Pathology-Reference-Cited by-同舟云学术

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Neuroinflammation-Modulating Agent SB1617 Enhances LC3-Associated Phagocytosis to Mitigate Tau Pathology

Published:2023-11-28 Issue:23 Volume:14 Page:4139-4152
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

Cho Hana¹,Choi Bo Young²³,Shin Young-Hee⁴⁵,Suh Sang Won²,Park Seung Bum¹⁴^ORCID

Affiliation:

1. Department of Biophysics and Chemical Biology, Seoul National University, Seoul 08826, Korea

2. Department of Physiology, College of Medicine, Hallym University, Chuncheon 24252, Korea

3. Department of Physical Education, College of Natural Sciences, Hallym University, Chuncheon 24252, Korea

4. CRI Center for Chemical Proteomics, Department of Chemistry, Seoul National University, Seoul 08826, Korea

5. Department of Chemical Engineering & Biotechnology, Tech University of Korea, Siheung 15073, Korea

Funder

National Research Foundation of Korea

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.3c00508

Reference53 articles.

1. The distribution of tau in the mammalian central nervous system.

2. Axonal Tau mRNA Localization Coincides with Tau Protein in Living Neuronal Cells and Depends on Axonal Targeting Signal

3. Sites of phosphorylation in tau and factors affecting their regulation

4. The Relationship between Amyloid and Tau

5. Self-propagation of pathogenic protein aggregates in neurodegenerative diseases

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